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Frontiers | New Strategies to Promote Macrophage Cholesterol Efflux
Frontiers | New Strategies to Promote Macrophage Cholesterol Efflux

Localization and lipid efflux of ABCA1, ABCG1, and ABCG4 on the plasma... |  Download Scientific Diagram
Localization and lipid efflux of ABCA1, ABCG1, and ABCG4 on the plasma... | Download Scientific Diagram

ABCA1 and ABCG1 as potential therapeutic targets for the prevention of  atherosclerosis - ScienceDirect
ABCA1 and ABCG1 as potential therapeutic targets for the prevention of atherosclerosis - ScienceDirect

Nutritionally Derived Metabolic Cues Typical of the Obese Microenvironment  Increase Cholesterol Efflux Capacity of Adipose Tissue Macrophages -  O'Reilly - 2019 - Molecular Nutrition & Food Research - Wiley Online Library
Nutritionally Derived Metabolic Cues Typical of the Obese Microenvironment Increase Cholesterol Efflux Capacity of Adipose Tissue Macrophages - O'Reilly - 2019 - Molecular Nutrition & Food Research - Wiley Online Library

Frontiers | Atrial Natriuretic Peptide Inhibited ABCA1/G1-dependent  Cholesterol Efflux Related to Low HDL-C in Hypertensive Pregnant Patients
Frontiers | Atrial Natriuretic Peptide Inhibited ABCA1/G1-dependent Cholesterol Efflux Related to Low HDL-C in Hypertensive Pregnant Patients

ABCG1 Antibody - BSA Free (NB400-132): Novus Biologicals
ABCG1 Antibody - BSA Free (NB400-132): Novus Biologicals

Extracellular lipid-free apolipoprotein E inhibits HCV replication and  induces ABCG1-dependent cholesterol efflux
Extracellular lipid-free apolipoprotein E inhibits HCV replication and induces ABCG1-dependent cholesterol efflux

JCDD | Free Full-Text | Genomic Variants and Multilevel Regulation of  ABCA1, ABCG1, and SCARB1 Expression in Atherogenesis
JCDD | Free Full-Text | Genomic Variants and Multilevel Regulation of ABCA1, ABCG1, and SCARB1 Expression in Atherogenesis

Cholesterol dyshomeostasis in amyotrophic lateral sclerosis: cause,  consequence, or epiphenomenon? - Hartmann - 2022 - The FEBS Journal - Wiley  Online Library
Cholesterol dyshomeostasis in amyotrophic lateral sclerosis: cause, consequence, or epiphenomenon? - Hartmann - 2022 - The FEBS Journal - Wiley Online Library

Brothers in Arms: ABCA1- and ABCG1-Mediated Cholesterol Efflux as Promising  Targets in Cardiovascular Disease Treatment | Pharmacological Reviews
Brothers in Arms: ABCA1- and ABCG1-Mediated Cholesterol Efflux as Promising Targets in Cardiovascular Disease Treatment | Pharmacological Reviews

Leonurine Prevents Atherosclerosis Via Promoting the Expression of ABCA1  and ABCG1 in a Pparγ/Lxrα Signaling Pathway- Dependen
Leonurine Prevents Atherosclerosis Via Promoting the Expression of ABCA1 and ABCG1 in a Pparγ/Lxrα Signaling Pathway- Dependen

Brothers in Arms: ABCA1- and ABCG1-Mediated Cholesterol Efflux as Promising  Targets in Cardiovascular Disease Treatment | Pharmacological Reviews
Brothers in Arms: ABCA1- and ABCG1-Mediated Cholesterol Efflux as Promising Targets in Cardiovascular Disease Treatment | Pharmacological Reviews

T cell cholesterol efflux suppresses apoptosis and senescence and increases  atherosclerosis in middle aged mice | Nature Communications
T cell cholesterol efflux suppresses apoptosis and senescence and increases atherosclerosis in middle aged mice | Nature Communications

Pro-apoptotic miRNA-128-2 modulates ABCA1, ABCG1 and RXRα expression and  cholesterol homeostasis | Cell Death & Disease
Pro-apoptotic miRNA-128-2 modulates ABCA1, ABCG1 and RXRα expression and cholesterol homeostasis | Cell Death & Disease

Role of HDL, ABCA1, and ABCG1 Transporters in Cholesterol Efflux and Immune  Responses | Arteriosclerosis, Thrombosis, and Vascular Biology
Role of HDL, ABCA1, and ABCG1 Transporters in Cholesterol Efflux and Immune Responses | Arteriosclerosis, Thrombosis, and Vascular Biology

ABCA1 and ABCG1 as potential therapeutic targets for the prevention of  atherosclerosis - ScienceDirect
ABCA1 and ABCG1 as potential therapeutic targets for the prevention of atherosclerosis - ScienceDirect

T-cell Abca1 and Abcg1 cholesterol efflux pathways suppress T-cell  apoptosis and senescence and increase atherosclerosis in middle-aged  Ldlr-/- mice | bioRxiv
T-cell Abca1 and Abcg1 cholesterol efflux pathways suppress T-cell apoptosis and senescence and increase atherosclerosis in middle-aged Ldlr-/- mice | bioRxiv

Brain‐specific loss of Abcg1 disturbs cholesterol metabolism and aggravates  pyroptosis and neurological deficits after traumatic brain injury - Xu -  2023 - Brain Pathology - Wiley Online Library
Brain‐specific loss of Abcg1 disturbs cholesterol metabolism and aggravates pyroptosis and neurological deficits after traumatic brain injury - Xu - 2023 - Brain Pathology - Wiley Online Library

ABCA1 and ABCG1 Synergize to Mediate Cholesterol Export to ApoA-I |  Arteriosclerosis, Thrombosis, and Vascular Biology
ABCA1 and ABCG1 Synergize to Mediate Cholesterol Export to ApoA-I | Arteriosclerosis, Thrombosis, and Vascular Biology

Sterol Efflux by ABCA1 and ABCG1 - ScienceDirect
Sterol Efflux by ABCA1 and ABCG1 - ScienceDirect

ABCA1 and ABCG1 as potential therapeutic targets for the prevention of  atherosclerosis - ScienceDirect
ABCA1 and ABCG1 as potential therapeutic targets for the prevention of atherosclerosis - ScienceDirect

JCDD | Free Full-Text | Genomic Variants and Multilevel Regulation of  ABCA1, ABCG1, and SCARB1 Expression in Atherogenesis
JCDD | Free Full-Text | Genomic Variants and Multilevel Regulation of ABCA1, ABCG1, and SCARB1 Expression in Atherogenesis

Evodiamine Lowers Blood Lipids by Up-Regulating the PPARγ/ABCG1 Pathway in  High-Fat-Diet-Fed Mice | Journal of Natural Products
Evodiamine Lowers Blood Lipids by Up-Regulating the PPARγ/ABCG1 Pathway in High-Fat-Diet-Fed Mice | Journal of Natural Products

Asprosin inhibits macrophage lipid accumulation and reduces atherosclerotic  burden by up-regulating ABCA1 and ABCG1 expression via the p38/Elk-1  pathway | Journal of Translational Medicine | Full Text
Asprosin inhibits macrophage lipid accumulation and reduces atherosclerotic burden by up-regulating ABCA1 and ABCG1 expression via the p38/Elk-1 pathway | Journal of Translational Medicine | Full Text